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Age and IQ were included as covariates of no interest in all analyses. Read Article at publisher’s site. May 5, Published: Academic Press, New York,pp. Abstract Gain spectra were calculated at critical wavelength degeneracy CWD in a collinear phase-matching geometry optical parametric amplification OPA process. Privacy Terms of Use. Right click equation to reveal menu options. Equations 17 Equations on this page are rendered with MathJax. Two fMRI contrasts were analyzed: Remember me on this computer.

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Europe PMC requires Javascript to function effectively. Third, the within-ASD analyses showed that a autism symptom severity but not disruptive behavior was uniquely associated with less activation in the social perception regions including the posterior superior temporal sulcus and inferior frontal gyrus; b disruptive behavior but not autism symptom severity was uniquely associated with less DMN deactivation in the medial prefrontal cortex MPFC and lateral parietal cortex; leei c anxiety symptoms mediated the link between disruptive behavior and less DMN deactivation in both anterior cingulate cortex ACC and MPFC, while ADHD symptoms mediated the link primarily in ACC.

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Eli 31 11 Express 16 4 Please login to set citation alerts. Express 22 17 The current research aims to better understand the neural underpinnings of disruptive behavior in ASD, while addressing whether the neural basis is shared with or separable from that of core ASD symptoms.

1169 Disruptive behavior in autism spectrum disorder ASD is an important clinical problem, but its neural basis remains poorly understood. Password Forgot your password? Citing articles from OSA journals and other participating publishers are listed here.

OSA will be closed for the holidays from 21 December We’ve also updated our Privacy Notice. March 29, Revised Manuscript: The current research aims to better understand the neural underpinnings of disruptive behavior in ASD, while addressing whether the neural basis is shared with or separable from that of core ASD symptoms.

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Two fMRI contrasts were analyzed: Click here to see what’s new. Click here to see a list of articles that cite this paper.

Equations on this page are rendered with MathJax. Gain spectra were calculated at critical wavelength degeneracy CWD in a collinear phase-matching geometry optical parametric amplification OPA process.

Click here to learn more. Dabu, “Very broad gain bandwidth parametric amplification in nonlinear crystals at critical wavelength degeneracy,” Opt.

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Express 17 15 Participants consisted of 48 male children and adolescents: In boys with ASD, disruptive behavior has a neural basis in reduced DMN deactivation, which is distinct and separable from that of core ASD symptoms, with the latter characterized by hypoactivation in the social perception circuitry. Age and IQ were included as covariates of no interest in all analyses. Third, the leii analyses showed that a autism symptom severity but not disruptive behavior was uniquely associated leii less activation in the social perception regions including the posterior superior temporal sulcus and inferior frontal gyrus; b disruptive behavior but not autism symptom severity was uniquely associated with less DMN deactivation in the medial prefrontal cortex MPFC and lateral parietal cortex; and c anxiety symptoms mediated the link between disruptive behavior and less DMN deactivation in both anterior cingulate cortex Lsi and MPFC, while ADHD symptoms mediated the link primarily in ACC.

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This website uses cookies to deliver some of our products and services as well as for ldi and to provide you a more personalized experience. April 30, Manuscript Accepted: Alert me when this article is cited. Participants consisted of 48 male children and adolescents: Find all citations in this journal default.

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Or filter your current search. United Crystals Company, Data sheets, http: How does Europe PMC derive its citations network? In boys with ASD, disruptive behavior has a neural basis in reduced DMN deactivation, which is distinct and separable from that of core ASD symptoms, with the latter characterized by hypoactivation in the social perception circuitry.